Inflammation of the mucosal lining of the stomach; may result from infection or excessive alcohol consumption. Atrophic gastritis is the progressive loss of gastric secretion with increasing age.
Inflammation of the stomach lining, with either congested and boggy membranes or inflamed membranes. It may be caused by bacteria and yeast, or chemical irritation like alcohol, but most frequently it is the result of emotional stress and inappropriate patterns of eating.
Inflammation of the stomach, causing damage to the blood vessels and erosion of stomach tissue.
Inflammation of the lining of the stomach, characterized by loss of appetite, nausea, vomiting, and discomfort after eating. Acute gastritis may be caused by the ingestion of an irritating substance (e.g., aspirin, too much alcohol) or by bacterial or viral infection; chronic gastritis is often a symptom of gastric ulcer, stomach cancer, pernicious anemia, or other disorder. Helicobacter pylori, a spiral-shaped bacterium found m the stomach, is generally acknowledged as the main cause for most peptic ulcers and many cases of chronic gastritis.
Inflammation of the mucous membrane lining the stomach. Gastritis may be sudden and acute, or chronic (persistent over a long period). In most cases, it exhibits no symptoms, but can cause indigestion, nausea, and vomiting. Gastritis is occasionally accompanied by gastric erosion, a superficial raw area in the mucous membrane that lines the stomach. Contributing factors can include bacteria, viruses, substances that damage the protective mucous lining of the stomach, the bacterium helicobacter pylori, alcohol use, heavy smoking, heavy use of nonsteroidal anti-inflammatory drugs (NSAIDs), or side effects of other drugs.
Inflammation of the lining (mucosa) of the stomach. Acute gastritis is caused by ingesting irritating or corrosive substances and causes vomiting. Chronic gastritis is associated with smoking and chronic irritation and may also be caused by bile entering the stomach from the duodenum. It has no definite symptoms, but the patient is liable to develop gastric ulcers. Atrophic gastritis, in which the stomach lining is atrophied, may succeed chronic gastritis but may occur spontaneously as an autoimmune disease. Dyspeptic symptoms such as nausea, vomiting, loss of appetite, and abdominal discomfort, popularly ascribed to gastritis, are not due to inflammation of the stomach.
Inflammation of the lining of the stomach. This may take an acute form when excess alcohol or other irritating substances have been ingested, resulting in vomiting. Chronic gastritis may be the result of regular smoking and chronic alcoholism; the condition may also be caused by the back-flow of bile from the duodenum. The common cause, however, is chronic infection with helicobacter pylori. Symptoms are vague, but sufferers are likely to develop gastric ulcers or sometimes cancer. Atrophic gastritis, when the mucosal lining of the stomach withers away, may follow chronic gastritis but sometimes occurs as an autoimmune disorder.
Acute or chronic inflammation of the lining of the stomach. Worldwide, the most common cause is infection with Helicobacter pylori. Other relatively common causes of gastric inflammation include use of alcohol and tobacco products and injury to the lining of the stomach by nonsteroidal anti-inflammatory drugs (NSAIDs). Autoimmune diseases (e.g., pernicious anemia); duodenal reflux; and gastric ischemia are sometimes responsible. Acute gastritis may develop in hospitalized patients (e.g., those with major traumatic injuries, burns, severe infections, organ failure, or major surgery).
The visceral mantle, located within our gastronomic cavity, experiences exacerbation as a repercussion of consistent vexation to its delicate infrastructure. The infliction, discernible in nature, can materialize in two divergent spectrums – it may unfurl as an abrupt, acute scenario, or insidiously embed itself as a relentless chronic predicament.
The onset of acute gastritis can frequently be attributed to the malevolent invasion of the Helicobacter Pylori microorganism. However, alternative catalysts also exist – one being pharmacological agents, predominantly those nonsteroidal anti-inflammatory entities like aspirin. Similarly, ethyl alcohol ingestion or strenuous physiological duress – exemplified by extensive burns or comprehensive surgical procedures – may trigger the malady.
The lingering variant of gastritis, more commonly termed as chronic, predominantly originates from a sustained H. Pylori onslaught. However, the etiology can also extend to persistent vexation brought about by factors like tobacco consumption, alcohol indulgence, or the caustic nature of bile. Equally, autoimmune anomalies causing deterioration to the gastric veneer, or the natural senescence that engenders degeneration of the mucosal layer, can play a substantial role.
The manifest symptoms commonly entail unease localized within the upper abdomen, accompanied by waves of nausea and episodes of emesis. In the acute variant of gastritis, excreta may assume a darkened hue, indicative of blood seepage from the stomach. Conversely, in its chronic manifestation, a gradual hemorrhagic process may give rise to anemic conditions. The resultant anemia then births a series of characteristic signs like a sallow complexion, persistent fatigue, and a heightened state of dyspnea.
The manifest symptoms commonly entail unease localized within the upper abdomen, accompanied by waves of nausea and episodes of emesis. In the acute variant of gastritis, excreta may assume a darkened hue, indicative of blood seepage from the stomach. Conversely, in its chronic manifestation, a gradual hemorrhagic process may give rise to anemic conditions. The resultant anemia then births a series of characteristic signs like a sallow complexion, persistent fatigue, and a heightened state of dyspnea.
The identification of gastritis relies significantly on the gastroscopy procedure – a method involving the exploration of the stomach’s interior using a flexible optical device. During this examination, a biopsy, the excision of tissue for further scrutiny, might be executed. The treatment plan may encompass ulcer-ameliorating pharmaceuticals and antibiotics specifically targeting the H. Pylori infection. Accompanying anemia will also be addressed with appropriate countermeasures. However, it’s worth noting that gastritis induced by an autoimmune disorder, regrettably, lacks a concrete therapeutic solution.
Swelling of the stomach’s inner lining.