A blockage of the pylorus, which prevents food from passing from the stomach into the duodenum.
Narrowing of the pyloric sphincter, blocking the passage of food into the duodenum from the stomach and often causing projectile (forceful) vomiting. It may be congenital or, in adults, result from an ulcer or neoplasm. Treatment is usually by surgery.
A rare disorder in which the pylorus, the outlet from the stomach to the duodenum, becomes partially or completely blocked. Pyloric stenosis is usually the result of a chronic peptic ulcer, which causes scarring and deformity of the outlet. Damage and blockage result from strong acids and digestive enzymes secreted by stomach glands. Less commonly, this condition is due to stomach cancer. Pyloric stenosis blocks the passage of food and secretions from the stomach to the duodenum. This often results in a bloated, uncomfortable feeling. Because the stomach is never fully emptied, the abdomen becomes distended. Vomiting may occur several hours or more after eating. Total blockage of the pylorus results in repeated vomiting, weight loss, dehydration, malnutrition, and chemical imbalances.
Narrowing of the muscular outlet of the stomach (pylorus). This causes delay in passage of the stomach contents to the duodenum, which leads to repeated vomiting (sometimes of food eaten more than 24 hours earlier), and sometimes visible distension and movement of the stomach. If the condition persists the patient loses weight, becomes dehydrated, and develops alkalosis.
Narrowing of the pylorus, the muscular exit from the stomach. It is usually the result of a pyloric ulcer or cancer near the exit of the stomach. Food is delayed when passing from the stomach to the duodenum and vomiting occurs. The stomach may become distended and peristalsis (muscular movement) may be seen through the abdominal wall. Unless surgically treated the patient will steadily deteriorate, losing weight, becoming dehydrated and developing alkalosis.
Narrowing of the pyloric orifice. In infants, excessive thickening of the pyloric sphincter or hypertrophy and hyperplasia of the mucosa and submucosa of the pylorus are usually responsible. In adults, obstruction of the pylorus usually results from peptic ulcer disease, malignant compression of the gastric outlet, or pneumatosis intestinalis.
A condition marked by the constriction of the pylorus (the lower opening of the stomach). Pyloric stenosis results in a blockage that hampers the flow of food into the duodenum, the initial section of the small intestine.
Pyloric stenosis develops in infants due to the thickening of the pyloric muscle. In adults, this condition arises from scarring caused by a peptic ulcer or stomach cancer.
Infants begin experiencing projectile vomiting, where stomach contents are forcefully expelled at a distance, around two to five weeks after birth. In adults with this condition, undigested food is vomited several hours after consuming a meal.
For infants, the diagnosis is confirmed through ultrasound scanning. In adults, however, the diagnosis is typically established using a barium X-ray examination and a gastroscopy, which involves using a viewing instrument to examine the stomach.
For infants, surgical treatment entails creating an incision along the thickened muscle. In adults, surgery is performed to address the root cause.
A blockage of the pylorus seen in infants, leading to severe vomiting. If not addressed, the child’s condition worsens due to the inability to consume fluids or food. Ramstedt’s operation is the solution for this condition.
A birth defect causing a blockage at the pyloric opening of the stomach, leading to persistent and forceful vomiting in infants. Typically, surgery to divide the pyloric muscle valve is the recommended solution.