Hemolytic anemia

Anemia due to hemolysis, the breakdown of red blood cells in the blood vessels or elsewhere in the body.


Disorder in which there is premature destruction of red blood cells. Anemia—abnormally low hemoglobin levels—may or may not be present, depending on the ability of bone marrow to increase red blood cells production. Hemolytic anemia can result from certain infections or inherited disorders of red blood cells but most often is a response to drugs or toxic substances (e.g., snake venom).


A group of diseases in which red blood cells are destroyed faster than they are produced, resulting in anemia. Symptoms include fatigue, pallid skin, breathlessness, rapid heartbeat (particularly on exertion), jaundice, dark urine, and enlarged spleen. Hemolytic anemias are sometimes difficult to treat, but they are rarely fatal.


Anemia as the result of the destruction of red blood cells (RBCs) by drugs, artificial heart valves, toxins, snake venoms, infections, and antibodies. Drugs may either destroy the RBC membrane directly or may stimulate production of autoantibodies that lyse (kill) the RBCs. Children may develop hemolytic anemia in response to destruction of RBCs by viral and bacterial organisms. Artificial valves cause physical damage to the RBC membrane during the circulation of blood through the heart.


A form of anemia characterized by the disintegration of red blood cells, resulting in the release of hemoglobin.


 


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